Professor Herbert Herzog
1986 Diplomprüfung Chemie - Mag. rer. nat. (Bachelor of Science equivalent)
University of Innsbruck, (Austria)
1989 Doktorprüfung - Dr. rer. nat. (Doctor of Philosophy equivalent)
Department of Biochemistry at the University of Innsbruck (Austria)
1996 Habilitation - Priv. Doz. (Doctor of Science equivalent)
Free University of Berlin, Department of Biochemistry
Prof Herzog studied Chemistry, switching to Biochemistry for his PhD, which he obtained from the University of Innsbruck (Austria) in 1989. In 1991, Prof Herzog moved to Sydney where he studies the role of NPY and other family members like PYY and pancreatic polypeptide, investigating the numerous different functions of these molecules in various physiological settings. Prof Herzog has published his findings on NPY and related neuropeptides in over 330 articles which are cited over 28000 times. He currently holdsa National Health and Medical Research Council of Australia Research Investigator (L3) grant.
- Publications
- Media
- Grants
- Awards
- Research Activities
- Engagement
- Teaching and Supervision
2024 – 2028 NHMRC Investigator Grant (L3)
NHMRC#2025606
Identification of neuronal networks driving combined HFD/chronic stress
acceleration of obesity development.
A$ 2,753.040,-
2021 – 2024 NHMRC Ideas Grant (CIA)
NHMRC# 2000617
Feeding behaviour and obesity development: Identification of novel intervention
points.
A$ 923.668,-
AWARDS AND HONOURS
1991 Erwin Schroedinger Fellowship (Austria)
2000 Wellcome Trust Short Term Travel Fellowship
2004 Barbara Ell Seminar Series Lecturer
2008 Victor Mutt Award (International Society for Regulatory Peptides)
2010 ANS Plenary Lecture
2017 Invitation Fellowship for Research in Japan
2023 Invitation Fellowship for Research in Japan
Prof Herzog’s current work focuses on determining the fundamental processes that can lead to the development of obesity, or the other extreme anorexia, especially investigating the brain's role in the regulation of eating behaviour, stress and glucose homeostasis. He is also interested in how homeostatic processes that regulate bodyweight are coordinated with other homeostatic processes in the body, like the one that control bone and fat mass and how these effects change with age.
To uncover the underlying mechanism that control the pathways that influence metabolism under stress conditions we have developed sophisticated transgenic mouse models which we comprehensively investigate via metabolic profiling in combination with AAV-viral tools (eg Cre- recombinase,Chemogenetic DREADD’s, Opto-genetic channels, TetTag systems, TRAP-seq and viral tracing, etc).